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Description
ACAT1-mediated ME2 acetylation drives chemoresistance in ovarian cancer by linking glutaminolysis to lactate production

This structural enhancement enables NAC to function as an effective prodrug that releases L-cysteine systematically after absorption, providing sustained amino acid availability for glutathione synthesis and other critical metabolic processes

SIQUEIRABAPTISTA, R

doi: 10.3389/fnbeh.2022.859151
