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It reduced cardiovascular mortality compared to placebo in patients with T2DM who were at high risk for cardiovascular events while on standard therapy [45]
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Alternatively, inhibiting pyruvate dehydrogenase kinase isoenzyme 1 (PDK1) with dichloroacetate (DCA) activates pyruvate dehydrogenase (PDH), channeling pyruvate into the tricarboxylic acid cycle instead of lactate production, mitigating extracellular matrix acidification [115,116,117]
it is a safety-critical design feature of the protocol
