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By upregulating beta-3 adrenergic receptors in adipose tissue, it increases the sensitivity of fat cells to lipolytic signals
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Recent studies also show that -sitosterol suppresses pro-survival signaling cascadesfor instance, an oxidized -sitosterol derivative significantly downregulated phosphorylated ERK1/2 and NF-B p65 levels, leading to increased ceramide accumulation and apoptosis in breast and liver cancer cells (97)
