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autophagy glutathione Glutathione-S-transferase omega 1 (GSTO1-1) acts as mediator of signaling pathways involved in aflatoxin B1-induced apoptosis–autophagy crosstalk in macrophages Antioxidant drugs inhibit basal and

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542 The safety of mitochondrial transplantation was evaluated in a clinical trial that included 30 patients with myocardial infarction, in which participants in the treatment group accepted autologous platelet-derived mitochondria injections into the coronary arteries, and after 40 days, the treatment group had improvements in left ventricular ejection fraction and exercise capacity, but there were no significant differences in the occurrence of adverse events such as arrhythmias and fever between the treatment and control group

autophagy glutathione Glutathione-S-transferase omega 1 (GSTO1-1) acts as mediator of signaling pathways involved in aflatoxin B1-induced apoptosisautophagy crosstalk in macrophages Antioxidant drugs inhibit basal and

The mice were injected with 4% thioglycollate medium intraperitoneally in order to isolate the peritoneal macrophages

autophagy glutathione Glutathione-S-transferase omega 1 (GSTO1-1) acts as mediator of signaling pathways involved in aflatoxin B1-induced apoptosisautophagy crosstalk in macrophages Antioxidant drugs inhibit basal and

Table 1 shows the group-specific demographics

autophagy glutathione Glutathione-S-transferase omega 1 (GSTO1-1) acts as mediator of signaling pathways involved in aflatoxin B1-induced apoptosisautophagy crosstalk in macrophages Antioxidant drugs inhibit basal and

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autophagy glutathione Glutathione-S-transferase omega 1 (GSTO1-1) acts as mediator of signaling pathways involved in aflatoxin B1-induced apoptosisautophagy crosstalk in macrophages Antioxidant drugs inhibit basal and

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