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Furthermore, the main physiological function of beta cells, glucose-stimulated insulin secretion (GSIS), is itself a significant source of oxidative stress [83]

245 Drugs exhibit poor stability in both in vivo and in vitro environments, making them prone to degradation or failure

Our previous studies have reported similar evidence in hepatocytes with low B12 demonstrating upregulation of FA and TG synthesis [14], and increased adiposity via hypomethylation and upregulation of the master regulators of lipogenesis (SREBF1) and cholesterologenesis (LDLR) in adipocytes [13]