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Embryos with optimal cleavage timing show coordinated expression of cell cycle genes and metabolic regulators, while delayed development correlates with altered gene expression patterns and metabolic dysfunction [433, 434]
However, increased p-FAK Tyr397 may be part of a mechanism that supports migration, or it may be a downstream consequence of altered adhesion, cytoskeletal tension, or cell-matrix interaction
TRIM29 and TRIM44 ( Table 1 ) are also reported to affect mTOR signaling (Xing et al., 2016
