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Description
The active-site cysteine residue of Ca(2+)/calmodulin-dependent protein kinase I is protected from irreversible modification via generation of polysulfidation
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While BPC-157 is noted to have a very short half-life (less than 30 min [51, 52]), the angiogenic, anti-inflammatory, and tissue regeneration processes it initiates appear to persist for weeks to months in animal studies, far exceeding the peptides pharmacokinetic presence [19, 53]
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