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Among them, oxidative stress leads to dysregulated production of adipokines, including plasminogen activator inhibitor-1 (PAI-1), tumour necrosis factor- (TNF-), monocyte chemoattractant protein-1 (MCP-1), and adiponectin, further reducing insulin sensitivity, impairing glucose and lipid metabolism, and early instigating metabolic syndrome [15]

This also suggests that copper homeostatic imbalance exacerbates ferroptosis, which in turn is involved in the induction of cuproptosis, which in turn leads to SAW (Figure 3)

Pentadecapeptide BPC 157 efficiently reduces radiation-induced liver injury and lipid accumulation through Kruppel-like factor 4 upregulation both in vivo and in vitro

e PLP, pyridoxal 5-phosphate