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Zhang G, Jiang P, Tang W, Wang Y, Qiu F, An J, et al
No improvement even after increasing to the maximum tolerated dose under medical supervision
described a redox-uptake system mediated by gliotoxin [55]: intracellular gliotoxin can be reduced to dithiol gliotoxin by a cellular reducing agent, thus releasing electrons to cellular O 2 to form O 2 -mediated gliotoxin, which can yield ROS
Pathogenesis of idiosyncratic drug-induced liver injury and clinical perspectives
