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In some cases, pathogenic mutations are observed in genes associated with the CoQ10 biosynthesis (primary CoQ10 deficiencies) or those not directly associated with CoQ10 biosynthesis (secondary CoQ10 deficiencies)

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Evidence from cultured cells showed that testosterone induced the levels of Pgc-1 and Tfam genes in C 2 C 12 myogenic cells (83) and increased the number of myotubular and mitochondrial DNA copies in myogenic cells, which was notably attenuated by an inhibitor of AR (84)

This variability degrades tail latency, also known as the 99th percentile response time or worst-case delays