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Zhu R, et al
Diabetes-associated excess kidney tissue RAS activation increases angiotensin II-mediated glomerular efferent arteriolar vasoconstriction and glomerular hyperfiltration, tubular sodium reabsorption, and signalling through intracellular pathways, resulting in inflammation and oxidative stress [13]
Example: A patient with CKD stage 4 , heart failure, and SIADH secondary to pneumonia may simultaneously have hypervolemic and euvolemic features, complicating both diagnosis and treatment
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