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This review critically examines the mechanistic, preclinical, epidemiological, and clinical evidence concerning the impact of semaglutide on cognitive function to reconcile these conflicting findings
I., Mnch A
A common crossroad is cellular senescence, a state of indefinite cessation of the cell cycle, triggered by mitotic stress, DNA damage, telomere erosion, mitochondrial dysfunction, oxidative stress, inflammation, mechanical stress and oncogenic activation [187]
