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These data in conjunction with multiple reports in the 1920s and 1930s that intestinal mucosa had hypoglycemic properties led the authors of both papers to hypothesize that a gut-derived humoral substance contributed to the regulation of insulin secretion (Figure 1)
GLP1-RAs address metabolic factors and systemic inflammation, while biologics specifically inhibit key inflammatory pathways like TNF- or IL-17
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doi: 10.1016/j.jaad.2018.12.070
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