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The neuroprotective properties of GLP-1 receptor agonists, consistent with our findings, have been repeatedly documente they restore neurovascular interactions and cerebral microcirculation in metabolic syndrome models (Estato et al
Similarly, exenatide restored mitochondrial morphology in db/db mice by reducing ROS accumulation and enhancing mitophagy [154]
The inclusion of additional treatment comparators, such as SGLT2is, and clinical parameters including HbA1c, would also be of interest
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