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Description
doi: 10.1038/nature14132 31
However, molecular mechanisms of how semaglutide may restore the balance of several interlinking aspects of metabolism to exert pleiotropic effects in obesity and diabetes complications are based mainly on preclinical data, while available evidence in humans appears to be lacking
Resistin is a key mediator of glucosedependent insulinotropic polypeptide (GIP) stimulation of lipoprotein lipase (LPL) activity in adipocytes
The same phenomenon was evident in those receiving other glucose-lowering medications (such as insulin or SGLT-2 inhibitors) as part of the standard of care (82, 85)
