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At the molecular level, semaglutide's mechanism of action involves the GLP-1 receptor (GLP-1R), a G-protein coupled receptor [8]

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There are a large number of studies that have reported the application of exendin-4, liraglutide and exenatide in the treatment of cognitive dysfunction and pathological features in AD models

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Many patients report changes in food preferences whilst taking these medications, prompting questions about whether GLP-1 reduces sugar cravings specifically

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