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Canadian Medical Association Journal
Intracerebroventricular infusion of neuropeptide Y increases glucose dependentinsulinotropic peptide secretion in the fasting conscious dog
reported that GLP-1-induced suppression of glucagon release is dependent of protein kinase A (PKA) and independent of glucose or paracrine effects mediated by insulin or by somatostatin acting on somatostatin receptor subtype-2 [16],[17]
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