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HMSC pretreated with Ex-4 or modified to express Exendin-4 (hMSC-Ex-4) showed reduced HO-induced apoptosis and increased phosphorylated AMPK levels, effects blocked by the AMPK inhibitor compound C [18]

Interestingly, these neurons were deactivated during consumption, comparable with the activation of DMH GLP-1R neurons during consumption to encode pre-ingestive satiation [48,61]