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The Boston Business Journal has more details here (heads up, there's a paywall)

One of the key features of neuropathic pain is central sensitization, where the CNS becomes hyper-responsive to pain stimuli [105]

Overall, the multimorbidity of depression and T2DM involves shared genetic risk factors and multiple underlying biological mechanisms, including HPA axis dysregulation, pro-inflammatory states, gut-brain axis interactions, and insulin signaling alterations, which can affect neurotransmission

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