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Our study demonstrated that NaB upregulated hepatic GLP-1R expression by inhibiting HDAC2, which improved hepatic GLP-1 sensitivity and prevented NAFL disease progression (Fig
Subsequently, mice with cardiomyocyte-specific (CM-specific) deletion of Glp1r also showed cardioprotective effects of liraglutide in an in vivo model of MI (22), suggesting that the cardioprotective effects of this agent are derived from actions independent of CMs and/or the GLP-1R
The rate varies based on starting weight, dosage, and concurrent lifestyle modifications
