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Moreover, oxidative stress can further impair p53 activity by suppressing its acetylation and increasing MDM2-mediated ubiquitin degradation
Nicotine, Carcinogen, and Toxin Exposure in Long-Term E-Cigarette and Nicotine Replacement Therapy Users
20 found that the expression of Ki67 decreased in the mouse brain aging model induced by D-galactose, and the corresponding aging degree could be slowed down after intervention with chrysin
We hypothesize that smoke exposure activates the parthanatos pathway in HBE cells and represents a cell death mechanism that contributes to smoking related lung diseases
