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Mechanistically, lanosterol administration increased the solubility of lens proteins and reduced oxidative stress by enhancing total antioxidant capacity and decreasing GSSG/GSH ratio in the lens cortex (150), and its effect was dependent on the severity of the condition or the lanosterol concentration in aqueous humor which varies from 0 to 31.61 ng/ml (150,153)
Notably, the key enzyme IDO produced by Kyn is the target gene of AhR, indicating that AhR is a central molecule mediating oxidative stress and affecting ferroptosis pathways (76)
doi: 10.1186/1471-2474-8-27
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