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Description
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Primary structure and expression of a functional human glucocorticoid receptor cDNA
The methionine overload would tend to decrease basal hyperhomocysteinemia rather than cause a further increase in basal hyperhomocysteinemia in patients bearing remethylation defects such as folate deficiency (associated or not with the C677T mutation of the MTHFR gene) (Durand et al, 1997a
This process culminates in the exertion of an oncogenic effect, the enhancement of glutamine metabolism, and the promotion of CRC cell proliferation [42]
