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Finally, prospects for ferroptosis-based interventions and future directions point to several promising treatment avenues, including regulating iron homeostasis via chelators or TfR1/FPN modulation, using antioxidants and lipid peroxidation inhibitors to target GPX4 and ACSL4, or employing gene therapies and stem cell/exosome approaches to combat ferroptosis

In patients with MASLD and obesity, the increased activity and expression of microsomal cytochrome P450 (CYP) 2E1 and CYP4A11 was observed, with this increase showing a significant correlation with elevated levels of circulating ketone bodies and FFAs, high-density lipoprotein, triglycerides, insulin, and insulin resistance (Aljomah et al

Thus, the data demonstrate the possibility of development of oxidative stress in quiescent center as a result of IAA accumulation in its cells