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Antibodies against DCN or AGER shielded Gpx4 knockout mice from cerulein-triggered pancreatitis, suggesting an indirect influence of ferroptosis-released DAMPs on immune cells [102]

Loss of p53 promotes the interaction between DPP4 and nicotinamide adenine dinucleotide phosphate oxidase 1 (NOX1), leading to the formation of the NOX1-DPP4 complex and mediating plasma membrane lipid peroxidation and ferroptosis [40]

On the other hand, it may be due to the dose-dependent biphasic effect of borneol that excessive concentrations may reduce permeability or cause barrier tightening [135]