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HAP1 mediates the axonal transport of BDNF, with prior studies demonstrating that HAP1 / mice display aberrant BDNF trafficking as indicated by alterations in BDNF distribution throughout the neuron and decreased activity-dependent release of proBDNF [18, 19]

We devised an endosome-escaping HLAH peptide by replacing K in KLAK peptide with H (Fig

is supported in part by a Banting and Best Diabetes Centre Novo Nordisk Chair in Incretin Biology, and a Sinai Health Novo Nordisk Foundation Fund in Regulatory Peptides

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