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The heritability of BD seems to obey a complex nonMendelian inheritance [18] and some risk variants have been identified in previous works [19]
Finally, amelioration of the G/F-actin ratio restores the disruption of the YAP signal induced by high glucose and upregulates the transcription factors Neurod1, Foxa1, and Foxa2, which are responsible for controlling L-cell differentiation [30] (Fig
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J., Millecamps M., et al
