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Description
COPPER DYSREGULATION RELATED TO ATP7B GENETIC ISSUES IN WILSON DISEASE Excessive hepatic copper deposition in Wilson disease results from impaired biliary excretion of excess copper due to dysfunction of the ATP7B gene [20,21,24,27]

Delivery of siRNA silencing P-gp in peptide-functionalized nanoparticles causes efflux modulation at the bloodbrain barrier

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People who participate in physical activities may require alteration regarding the duration of their exercise or moments when they are physically exerting themselves
