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Studies show that the XPC variants are associated with increased DNA adduct levels after aflatoxin B1 exposure, increasing liver cancer risk.[ref][ref][ref] This means that for people who both accumulate more DNA damage from mycotoxins (because of detox variants) and repair it less efficiently (because of XPC variants), the longterm cancer risk from chronic exposure is significantly higher

However, the seventh (ADH6) has not been isolated as a protein in vivo, and its role in ethanol metabolism is still unknown (113) (Table 1)

Fourth, combination therapy: co-delivery systems (e.g., lipid-based vesicles co-encapsulating anti-inflammatory and urate-lowering agents) synergistically address inflammation and hyperuricemia, simplifying treatment regimens (Li et al., 2023)