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They also support the formation of new blood cells, delivering oxygen and nutrients to damaged areas
doi: 10.3390/nu12030792
70 To safely restore the functions of the GI epithelial barrier and protect against cytokine-dependent, MLCK-mediated TJ disruption, a recent study has disclosed the non-enzymatic interruption of MLCK recruitment to perijunctional actomyosin rings as a novel therapeutic
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