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In support of this, in a dual-hit genetic-environmental mouse model of SCZ, where DISC1 mutation was combined with MIA, transient administration of minocycline, an anti-inflammatory antibiotic drug, rescued electrophysiological and structural deficits during early postnatal development, as well as cognitive abilities in juvenile mice (Chini et al., 2020)

Bioinformatic analysis revealed insulin signaling pathway key molecules being affected in AD patients of which the six kinases, leucine-rich repeat kinase 2 (LRRK2), glycogen synthase kinase 3 beta (GSK3B), AKT serine/threonine kinase 1 (AKT1), epidermal growth factor receptor (EGFR), mitogen-activated protein kinase 1 (MAPK1), and the non-receptor tyrosine kinase FYN, played central roles [56]

Training method The EX and EXIN groups were adapted to treadmill training intensity of 12 m/min, 8 slope for 3 days