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Additionally, there was variability between the included studies in the follow-up duration, which may cause heterogeneity in the results obtained
We thus hypothesized that CD38 upregulation might have a fundamental pathogenic role in SSc fibrosis, and further, restoring NAD + homeostasis will prevent the process
Moreover, myofibroblast activation and the development of multiple organ fibrosis are substantially attenuated in CD38-null mice with elevated NAD + 24
