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The fact that oxaliplatin is more efficient than cisplatin could be explained by the fact that DNA-oxaliplatin adducts are more effective in inhibiting DNA synthesis and less susceptible to the repair mechanisms of the cell (Dilruba and Kalayda, 2016

J Mol Cell Cardiol 28:667678

Since all consumed methylations of every single substance participating in the one-carbon transfer must be recycled by itself, it is not sufficient to examine only the folate, sulfate and homocysteine pathway, but every individual regeneration pathway of each single coenzyme involved must be integrated into the overall consideration

At the late stage, increased lipophagy releases PUFAs that serve as substrates for lipid peroxidation, whereas the knockdown of RAB7A or PGRMC1 inhibits lipophagy-induced ferroptosis [210, 211]