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The following experimental details from each study are presented in Table 3: cancer type, chemotherapy agent tested, cell models used (divided by chemosensitive and chemoresistant cell lines), method of MET/HGF blockade employed, observed changes in MET/HGF leading to enhanced MET signalling and the MET-dependent mechanisms of chemotherapy resistance

doi: 10.1210/jc.2004-2190

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2224 Advances in genome annotation, enzyme localization, and definition of available substrates have allowed for a more fine-tuned prediction of essential properties and metabolic activities within Plasmodium in recent years