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Funding This work was supported by the National Institutes of Health (NIH)s National Cancer Institute (NCI) Grants 5F31CA275336-02, R01CA271597, and 5R01CA232589, and the Lineberger Comprehensive Cancer Centers Center for Triple Negative Breast Cancer
Additionally, the regulatory properties of GST in kinase cascades have provided a translational opportunity to target GSTs in myeloproliferative pathways, with the consequent clinical testing of new agents in myelodysplastic syndrome
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