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Receptors Conceptually, neurotransmitter activity, as the basis for the cytoprotective effects of the implemented agents, was not in the implementation of the cytoprotection concept since seminal studies [16,17,18,19,20,21]

doi: 10.1007/s12011-008-8117-8

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demonstrated that oxidative stress and H 2 O 2 treatment led to a marked increase in senescence-associated -galactosidase activity but only to minimal apoptotic cell death in CSCs, suggesting that ROS triggered the induction of senescence and exhibited therapeutic potential in the eradication of drug-resistant CSCs [8]