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Description
Roles of necrosis, apoptosis, and mitochondrial dysfunction in S-(1,2-dichlorovinyl)-L-cysteine sulfoxide-induced cytotoxicity in primary cultures of human renal proximal tubular cells
^ Hepatocyte-specific Gclc deletion leads to rapid onset of steatosis with mitochondrial injury and liver failure
The biological half-life of FB1 was estimated to be slightly under 48 h for short-term exposure (3 days) and between 48 and 72 h for longer exposure periods (6 days) (Riley et al
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