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This could be due to ALCs quick absorption and capacity to cross the blood-brain barrier, potentially altering the gut microbiota composition and limiting the abundance of TMA-producing bacteria
doi: 10.1007/s11064-007-9343-8
[13] Metabolic defects of fatty acid oxidation [edit] More than 20 human genetic defects in fatty acid transport or oxidation have been identified
In contrast, oral supplementation with acetylcarnitine for one month in old rats does not increase the total energy expenditure, despite the increase in fat utilization as metabolic fuel [99]
