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p21 and glutathione A redox switch in p21-CDK feedback during G2 phase controls the proliferation-cell cycle exit decision: Molecular Cell and Rbx1/Cul4B/Dcaf11/Ddb1 complex (E3) for 384 reactions The p53-p21 pathway suppresses ferroptosis

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10.1016/j.jamda.2024.03.007 45

p21 and glutathione A redox switch in p21-CDK feedback during G2 phase controls the proliferation-cell cycle exit decision: Molecular Cell and Rbx1/Cul4B/Dcaf11/Ddb1 complex (E3) for 384 reactions The p53-p21 pathway suppresses ferroptosis

The GCL enzyme is a heterodimer formed by two subunits: the heavy subunit or glutamate cysteine ligase catalytic subunit (GCLC, 73 kDa) and the light subunit or glutamate cysteine ligase modulating subunit (GCLM, 30 kDa)

p21 and glutathione A redox switch in p21-CDK feedback during G2 phase controls the proliferation-cell cycle exit decision: Molecular Cell and Rbx1/Cul4B/Dcaf11/Ddb1 complex (E3) for 384 reactions The p53-p21 pathway suppresses ferroptosis

A meta-analysis comparing the thermal pulsation system (LipiFlow) with control interventions (including placebo, warm compresses, and other thermostatic devices) revealed no statistically significant improvement in OSDI scores

p21 and glutathione A redox switch in p21-CDK feedback during G2 phase controls the proliferation-cell cycle exit decision: Molecular Cell and Rbx1/Cul4B/Dcaf11/Ddb1 complex (E3) for 384 reactions The p53-p21 pathway suppresses ferroptosis

Mitochondrial Dysfunction, Oxidative Stress, and Neuroinflammation: Intertwined Roads to Neurodegeneration

p21 and glutathione A redox switch in p21-CDK feedback during G2 phase controls the proliferation-cell cycle exit decision: Molecular Cell and Rbx1/Cul4B/Dcaf11/Ddb1 complex (E3) for 384 reactions The p53-p21 pathway suppresses ferroptosis

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