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Selective c-Met inhibitors have a very high selectivity (more than ten thousand times) for c-Met compared to other kinases [155]

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This may be a physiological role (in situ hybridization and immunostaining BPC 157 in human gastrointestinal mucosa, lung bronchial epithelium, epidermal layer of the skin and kidney glomeruli), and BPC 157 may act as stabilizer of cellular junction, leading to significantly mitigated leaky gut syndrome, via increasing tight junction protein ZO-1 expression, and transepithelial resistance [19]

To address this research gap, we conducted this study in which we measured shear modulus values to evaluate PF elasticity