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Ruemmele FM, Beaulieu JF, Dionne S et al (2002) Lipopolysaccharide modulation of normal enterocyte turnover by toll-like receptors is mediated by endogenously produced tumour necrosis factor alpha
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(2007) found that the activity of trypsin/ -chymotrypsin on proteins decreased as the carbonylation level in the latter increased, which provides strength to the role of protein carbonylation on the impaired digestibility of oxidized proteins
Phase 3 clinical data demonstrated superior weight reduction compared to GLP-1 monotherapy