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Therefore, the priority is to identify new autoantibodies against the neuronal surface antigens [114]
Based on overall FISH and Hi-C results, our model is that domain-based chromosomal compaction is diminished in the cnd1-K658E mutant, whereas domain compaction is partially compromised in Pmc4-depleted cells, coinciding with impaired boundary formation (Fig
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Dosages and stacking protocols.
