cancer and glutathione in Cell Death A study shows that the

Transcription profiles of ESCs following erastin-induced ferroptosis in vitro To explore why endometrial cells of ectopic lesions survive and grow in spite of stromal cell ferroptosis and also if ESC ferroptosis benefits the progression of ectopic lesions, we investigated erastin-induced ESC ferroptosis

Perhaps exemplifying this point, inhibition of the cystine/glutamate exchanger results in activation of an integrated stress response (but probably not a bona fide ER stress response) in cancer cells (Dixon et al., 2014), and this response is activated by, among other stimuli, amino acid depletion (Donnelly et al., 2013)

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Improved angiogenesis-related pathway activation