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The stability of xCT is regulated by the MUC1-C (Mucin 1 C-terminal subunit)/CD44v (CD44 variant) complex, which directly interacts with xCT to promote its stability and control GSH levels[122]

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Therefore, it is impossible to determine if IN GHK-Cu actually restored the aged hippocampus to a youthful transcriptomic state, or if it pushed the tissue into a novel, alternative compensatory state that simply happens to function better

doi: 10.1080/19338244.2018.1492894 273 Tayri-WilkT.SlavinM.ZamelJ.BlassA.CohenS.MotzikA.et al