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Mitochondrial failure in cells could increase H4K8 acetylation and accelerate telomere depletion, ultimately leading to an age-dependent decline in cell function [74, 75]

PKUMDL-LC-102 formed a water bridge due to the following protein residues: Asp21, Asp23, Asp30, Lys31, Arg33, Asp58, His60, Arg62, Tyr63, Ala64, Glu65, Cys66, Leu68, Arg64, Lle70, Val98, Lys99, Phe100, Asp101, Pro124, Lys125, Lys127, Gly128, Arg152, Glu163, Lys164, and Phe170

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