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During the initial meeting, the premise was identified and the overall methodology was defined
Alzheimers disease therapeutics: Current strategies and future directions
Although initial studies suggested that GSH depletion was only a byproduct of oxidative stress generated during cell death, recent discoveries suggest that GSH depletion and post-translational modifications of proteins through glutathionylation are critical regulators of apoptosis
In some experiments on spontaneously hypertensive rats, it has been found that catalpol treatment upregulates several regulatory proteins during prefrontal cortex development [152]
