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When the capability of glucose metabolism to produce ATP is impaired in normally proliferating cells, they undergo cell cycle arrest and reactivate catabolism while activating signaling pathways, such as AMP-activated protein kinase (AMPK) and activating protein kinase B (AKT), to maintain energy homeostasis ( 2 to maximize ATP generation, in conflict with the requirements of growing cells

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Ferritin: structure, biosynthesis, and role in iron metabolism