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2013 Sep 15;191(6):3419-29
& de Carvalho, L
Therefore, the reestablishing of blood supply in reperfusion phase enhances several mechanisms that are more detrimental for renal parenchyma and include the burst of ROS, calcium overload, opening of the MPT pore, endothelial dysfunction and activation of coagulation and inflammatory response (88, 89)
Histone citrullinase PAD4 is a key mediator of NET formation and its inhibitors (YW3-56 and YW4-03) can markedly reduce liver injury in HIRI mice (40)
