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Interestingly, inhibition of NLRP3 inflammasome activation reversed the M1 polarization of TMAO-stimulated macrophages, suggesting that NLRP3 plays a crucial role as a proteolytic activator in TMAO-induced macrophage responses
Conclusions KD demonstrates therapeutic potential in the management of a broad range of metabolic and neurological diseases
Creatina etil ster , 8
