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Despite the promising findings summarized in this review, several limitations should be acknowledged
doi: 10.1161/01.cir.86.4.1302
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reported that GLS-driven Gln metabolism is a regulator of radiotherapy tolerance in prostate cancer (PCa) and that high expression of GLS 1 and c-MYC, key regulators of Gln, are significantly associated with reduced progression-free survival in prostate cancer patients treated with radiotherapy
